POWERPOINT DECOMPRESSOR FULL

(iii) Full three wall decompression (lateral and medial walls, and orbital floor, usually reserved for severe degrees of proptosis) – Additional risks to (i) and (ii) above: In addition to the above, a medial wall decompression carries the following specific risks:

(ii) Balanced two wall decompression (lateral and medial walls): New onset double vision is very rare with a lateral wall decompression.

Swelling of the upper and lower eyelids, this settling over a few weeks.Ī ‘wobble’ of the vision on eating (‘masticatory oscillopsia’) – this occurring in about 40% of patients, but rapidly settling in nearly all. Orbital decompression is major surgery, and should only be considered after careful discussion with your ophthalmologist and with an understanding of all the associated risks, which are outlined below.Ī fine linear scar hidden within the ‘laughter lines’ of the outer corner of the eyelids. What are the risks of orbital decompression surgery? This frequently corrects the down-ward displacement of the lower lid and some of the bulging of the upper eyelid, although upper lid retraction may even increase and requires subsequent lid lowering In the surgical rehabilitation of patients with long-standing TED, decompression is performed to reduce proptosis. Thus, the main reasons for orbital decompression are (i) to reduce proptosis and (ii) to treat visual loss due to raised intraorbital pressure. Depending on the degree of proptosis, surgery may involve removing part of one, two or three orbital walls. The operation is achieved either by removing part of the wall(s) of the orbit, and, in some patients, by the additional excision of some of the orbital fatty tissue. Orbital decompression, typically performed for patients with proptosis in thyroid eye disease (TED), increases the available space for the orbital contents, thereby reducing the degree of proptosis. What is the purpose of orbital decompression surgery? Thus, the soft tissues of the orbit lie within a protected, but relatively confined, space, and expansion of these tissues (for example muscle enlargement in thyroid eye disease) can lead to ‘bulging’ of the eyes (proptosis) and rarely visual loss due to compression of the optic nerve at the apex of the orbit. The structures which enter the orbit from the brain do so via various holes in the bones behind the eye. The temporalis muscle, which closes the jaw, passes under the outer bony arch of the upper cheek (the zygoma) and lies against the bony wall of the outer aspect of the orbit. In the midline, lying between the two orbits, are the ethmoid sinuses and nasal space. Above are the frontal sinuses and brain, and below the air-filled maxillary sinuses. The eye, its surrounding muscles, nerves and fatty tissue lie within the bony orbit of the skull.

Powerpoint decompressor